By Icon Health Publications
It is a 3-in-1 reference ebook. It provides an entire clinical dictionary protecting hundreds and hundreds of phrases and expressions in terms of bilirubin. It additionally provides vast lists of bibliographic citations. eventually, it offers details to clients on tips to replace their wisdom utilizing numerous web assets. The ebook is designed for physicians, clinical scholars getting ready for Board examinations, scientific researchers, and sufferers who are looking to get to grips with learn devoted to bilirubin. in case your time is effective, this booklet is for you. First, you won't waste time looking out the net whereas lacking loads of proper info. moment, the booklet additionally saves you time indexing and defining entries. eventually, you won't waste time and cash printing 1000's of websites.
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Additional info for Bilirubin - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References
HO-1 catalyzes the breakdown of heme leading to the formation of the vasodilator CO and the antioxidant, bilirubin. Like NO, CO activates guanylyl cyclase resulting in elevated cGMP levels which in turn cause VSMC relaxation and limit VSMC proliferation. Our data show that it is the increase in CO by VSMC that (1) inhibits the hypoxic induction of the endothelial cell-derived mitogens, endothelin and platelet derived growth factor-B, indirectly inhibiting VSMC proliferation; (2) that CO also suppresses VSMC proliferation independent of endothelial cells by inhibiting the expression of E2F-1, a cell cycle-specific transcription factor; and (3) inhibits the trans-activating function of hypoxia-inducible factor-1 on gene expression by hypoxia.
Cholestatic liver injury results in a substantial clinical burden leading to an estimated 100,000 deaths per year often listed as multiple organ dysfunction. The current clinical management of chotestasis includes phenobarbital, which decreases hepatotoxicity as a side effect of cholestasis. I have previously demonstrated that treatment with multiple reducers of CYP2B 1/2, (transcriptionally activated by CAR) such as phenobarbital, as well as inducers of NADP(H):quinone oxidoreductase (activated through Nrf2) are also capable of inducing Mrp3, showing coordinate regulation of both the Phase I drug-metabolizing genes and Phase III xenobiotic transporters.
Surgery; University of Washington Grant & Contract Services Seattle, Wa 98105 Timing: Fiscal Year 2003; Project Start 01-MAR-2003; Project End 28-FEB-2005 Studies 27 Summary: (provided by applicant): Transduction of enterocytes with gene products may be used to augment or replace defective enzymes that are involved in absorption, secretion, and digestion. The main hypothesis is that viral transduction of intestinal stem cells with wild-type gene product will lower bilirubin levels in Gunn Rats that are deficient for bilirubin UDP-glucuronosyltransferase.
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